Mechanical Back Strain

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Continuing Education Activity

Mechanical back strain is a prevalent disease in the medical field. Most patients who develop a mechanical back strain have a benign course; however, some will continue to have chronic, disabling symptoms. While evaluation and management are usually conservative for mechanical back strains, healthcare providers must differentiate between other causes of back pain and be aware of when to search for other life-threatening etiologies. This activity reviews the etiology, epidemiology, evaluation, differential diagnoses, and treatment of mechanical back strains. It highlights the role of an interprofessional team in the treatment of patients with mechanical back strain.


  • Describe the etiology of mechanical back strain.

  • Outline the appropriate history, physical, and diagnostic evaluation of mechanical back strain.

  • Summarize the current recommendations for pharmacologic, non-pharmacologic, and invasive treatment options available for mechanical back strain.

  • Explain the importance of interprofessional team strategies for improving care coordination to advance the management and improve outcomes in patients with a mechanical back strain.


Back pain is one of the most common chief complaints in the emergency department. It is also a leading cause of disability and high healthcare expenses globally, as it affects patients of all ages and backgrounds. Mechanical back strain is a subtype of back pain where the etiology is the spine, intervertebral discs, or the surrounding soft tissues. While the majority of cases of mechanical back strains that present to the emergency department can be managed conservatively, healthcare providers must be aware of the common red flags that signal more emergent causes of back pain and that are associated with high morbidity and mortality when the diagnosis is delayed.[1][2]


Historically, references to mechanical back pain have used many other vague terms like "non-specific back pain," "lumbar strain," or "lumbago." This terminology was due to the absence of proper diagnostic methods in the past. However, more recent advances in imaging technology and a better clinical understanding of spinal anatomy and the body's innervation have allowed more accurate terminology and diagnoses, depending on the underlying cause. Hence, more specific diagnoses are made based on the etiology of mechanical back strain, which is typically due to pathology involving the spine's anatomy. While the lower back is usually affected the most, mechanical back pathology may involve any part of the bones, spinal ligaments, intervertebral discs, facet joints, spinal cord, spinal nerves, or paraspinal muscles. Common examples of pathology involving these structures include the following diseases[3]

  • Spinal foraminal stenosis
  • Herniated discs
  • Spinal stenosis
  • Degenerative disc disease
  • Vertebral fractures
  • Sacroiliac joint dysfunction
  • Facet joint syndrome
  • Ligamentous and muscular injury
  • Myofascial pain syndrome


Greater than 80% of people will suffer from low back pain during their lifetime. The global point prevalence of low back pain is 12% to 33%. There is a higher prevalence among women and people ages 40 to 80. Due to missed workdays and decreased wages, 100 to 200 billion dollars are estimated to be spent each year in the United States due to lower back pain.[2][4]

History and Physical

Obtaining a complete history and performing an in-depth physical exam is crucial to diagnosing mechanical back strains. The goal of evaluation in the acute setting is to ensure no red flags warrant more advanced imaging and surgical consultation. These red flag symptoms include fevers, involuntary weight loss, night sweats, bladder or bowel incontinence, significant trauma, frequent falls, history of cancer, anticoagulant use, chronic systemic steroid use, intravenous drug abuse, or an immunocompromised state. Physical exam red flags compromise assessing focal neurologic deficits, including weakness, ataxia, saddle anesthesia, decreased deep tendon reflexes and diminished rectal tone.[3][5]

Acute mechanical back strains may be triggered by physical or non-physical activity, with lifting being the most commonly recalled event. However, one-third of patients may not necessarily remember an inciting incident. More specific spinal pathology may have classically associated symptoms. For example, herniated discs will present with radicular pain that radiates from the lower back in L4 to S1 distribution, and pain may be reproducible to the affected side on straight leg raise. Spinal stenosis may demonstrate back pain and bilateral posterior leg pain that worsens with ambulation and spine extension but is relieved by rest and spine flexion. Patients on chronic steroids or at risk for osteoporosis are at increased risk for vertebral compression fractures even after seemingly minor trauma. Compression fractures tend to be focally tender on palpation.[1][6]


In the absence of the red flags discussed above, no laboratory or radiographic studies are necessary to diagnose or manage mechanical back strains in the acute setting. Inflammatory biomarkers, such as erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP), are useful for risk stratification of patients with risk factors for infectious spinal pathology or malignancy but have no neurologic deficits on examination. Routine imaging for mechanical back strains is not recommended, as many may have incidental abnormal findings that are unrelated to their pain. More advanced imaging is needed in trauma, failure of conservative management, worsening of symptoms, and new neurologic deficits. Plain radiographs and computed tomography are useful when suspecting fractures. However, patients with signs of spinal cord compression or red flag symptoms need magnetic resonance imaging for an accurate diagnosis.[3][5][7]

Treatment / Management

Management of mechanical back strains depends on the chronicity of symptoms, the patient's comorbidities, and the specific etiology. The American College of Physicians published an updated guideline in 2017 with recommendations regarding non-invasive options for treating low back pain. First-line nonpharmacologic therapy for acute low back pain includes spinal manipulation, acupuncture, massage, and superficial heat application, while first-line pharmacologic therapy for acute low back pain is nonsteroidal anti-inflammatory drugs and muscle relaxants. According to low-quality evidence, the use of acetaminophen and systemic corticosteroids showed no improvement of acute back pain compared to placebo. There was insufficient evidence to determine the benefits of other pharmacologic agents, such as benzodiazepines, antidepressants, antiepileptic medications, and opioids. According to the clinical policy by the American College of Emergency Physicians, opioids should not be routine pharmaceutical therapy but should be saved for those whose pain is severe or uncontrolled with other medications.[8][9]

Physical therapy is an integral component in the treatment of mechanical back pain. When initial conservative measures fail, a referral is appropriate. Studies have shown that early physical therapy intervention, when compared to the implementation of physical therapy after lower back pain was chronic, resulting in a reduction in healthcare costs, a decrease in opioid use, and a reduction in spinal surgery rates.[10][11]

For chronic low back pain, non-pharmacologic approaches were recommended as the first-line agents, including exercise, tai-chi, yoga, multidisciplinary rehabilitation, spinal manipulation, acupuncture, psychotherapy, low-level laser therapy, and electromyogram biofeedback. Nonsteroidal anti-inflammatory drugs were again the first-line pharmacologic agents recommended, followed by tramadol and duloxetine as the second-line treatments. Recommendations for opioid therapy are only if the previously mentioned therapies failed and based on an individualized decision to determine if the benefit outweighs the risk.[8]

The American Pain Society also published recommendations by a multidisciplinary panel in 2009 regarding the use of more invasive procedures for diagnostic and therapeutic purposes to treat non-radicular low back pain, radicular low back pain, and spinal stenosis. The panel emphasized the use of intensive interdisciplinary rehabilitation for chronic non-radicular low back pain, as it has similar efficacy to fusion surgery. Epidural steroid injection showed variable evidence of short-term benefit for radicular low back pain; thus, the panel recommended a shared decision-making process for the appropriate patient.

For surgical management options, standard open discectomy and microdiscectomy have a moderate short-term benefit in treating persistent, debilitating radiculopathy secondary to herniated disc disease. Similarly, decompressive laminectomy has a moderate short-term benefit in treating severe, debilitating spinal stenosis. The panel noted that these benefits tend to decrease over time, and a shared decision-making process is also appropriate in these patients before surgical intervention. Lastly, spinal cord stimulation may be an option for patients who underwent surgery for herniated disc disease but continue to have debilitating radiculopathy.[12]

Differential Diagnosis

The differential diagnoses for mechanical back strains are other nonmechanical causes of back pain. These include but are not limited to the following categories and diseases[1][5][13]

  • Rheumatologic - ankylosing spondylitis, Reiter syndrome, psoriatic spondylitis, polymyalgia rheumatica   
  • Oncologic - metastatic disease, spinal cord tumor, lymphomas, leukemia, multiple myeloma 
  • Infectious - spinal epidural abscess, osteomyelitis, discitis 
  • Gastrointestinal - pancreatitis, cholecystitis, bowel perforation 
  • Vascular - aortic aneurysm, spinal epidural hematoma, aortoiliac disease
  • Renal - pyelonephritis, nephrolithiasis, perinephric abscess
  • Genitourinary - endometriosis, prostatitis, pelvic inflammatory disease


While most acute episodes of mechanical back pain resolve spontaneously within 12 weeks, up to one-third of patients may continue to have chronic symptoms. However, even those whose acute symptoms resolve have a 20 to 40% chance of symptom recurrence during the first year and 85% of lifetime recurrence.[2][8] Various environmental and psychosocial factors may influence the prognosis of mechanical back strains. These include a body mass index greater than 25, smoking, poor health, depression, fear avoidance, strenuous work demands, job dissatisfaction, and the presence of compensation.[1][3][6]


The possible complications of mechanical back strains depend on the specific etiology, with the most common complication being the persistence of symptoms and the development of lasting disability. Rarer physical complications include cauda equina syndrome and physical deformities. Other health complications may arise as side effects from long-term pharmacologic therapies, such as NSAID use, or more invasive therapeutic procedures, such as injections and surgeries. Many financial and social complications also exist for both the patients with chronic, disabling mechanical back pain and their employers due to lost wages and missed workdays.[1][8]

5% to 10% of adults with low back pain develop chronic pain. Recurrence of low back pain occurs in 20 to 72% of patients. In adults with low back pain severe enough to cause them to miss work, 60 to 70% return to work by 6 weeks, and 80 to 90% return by 12 weeks.

Deterrence and Patient Education

Reduction of strenuous physical requirements is the main primary prevention for mechanical back strains; however, secondary prevention rather than primary prevention is more practical due to the high prevalence of the condition. The greatest risk factor is the recurrence of a previous episode. Some interventions by employers that may aid in injury prevention are stretching exercises at the workplace, appropriate rest breaks, and ergonomic modifications. Ergonomic modifications refer to adaptations in the work environment to reduce the physical stress of the employees. Also, educating patients regarding the importance of maintaining proper posture and correct lifting techniques may aid in prevention. Other prevention techniques that patients can use aim to reduce the risk factors of having a poor prognosis, including exercising, smoking cessation, weight loss for obese patients, and resuming normal physical activity.[1][14][15][16]

Enhancing Healthcare Team Outcomes

Healthcare professionals of many specialties are involved in providing care for patients with mechanical back strains. Physicians and nurses, especially in the acute setting, must be familiar with red flag symptoms of back pain that may require more urgent interventions and possible surgical consultation. Conservative management of acute mechanical back strains after a complete assessment is reasonable as the course is usually self-limited; this includes superficial heat, nonsteroidal anti-inflammatory drugs, and muscle relaxants.[8] [Level 2] 

Chronic mechanical back strains require an interprofessional team approach due to the psychosocial factors involved. Initial non-pharmacologic therapy includes exercise, interprofessional rehabilitation, manipulation, and acupuncture. First-line pharmacologic therapy is nonsteroidal anti-inflammatory drugs followed by duloxetine or tramadol as second-line therapy.[8] [Level 2] A pharmacist should review the patient's medications to ensure there are no drug interactions and that dosing is appropriate. Patients with persistent disabling chronic non-radicular low back pain may benefit from interprofessional rehabilitation, described as rehabilitation along with a psychological, social, or occupational component. Here is where an orthopedic specialty nurse can help coordinate therapy, monitor progress, answer patient questions, and inform the managing clinician if further intervention may be necessary. Patients with persistent chronic radicular pain due to herniated discs or spinal stenosis may benefit from surgical interventions.[12] [Level 1] 

Mechanical back pain requires an interprofessional team approach, including physicians, specialists, specialty-trained nurses, physical therapists, chiropractors, and pharmacists, collaborating across disciplines to achieve optimal patient results. [Level 5]



8/8/2023 1:53:33 AM



Casiano VE, Sarwan G, Dydyk AM, Varacallo M. Back Pain. StatPearls. 2023 Jan:():     [PubMed PMID: 30844200]


Freburger JK, Holmes GM, Agans RP, Jackman AM, Darter JD, Wallace AS, Castel LD, Kalsbeek WD, Carey TS. The rising prevalence of chronic low back pain. Archives of internal medicine. 2009 Feb 9:169(3):251-8. doi: 10.1001/archinternmed.2008.543. Epub     [PubMed PMID: 19204216]


Chien JJ, Bajwa ZH. What is mechanical back pain and how best to treat it? Current pain and headache reports. 2008 Dec:12(6):406-11     [PubMed PMID: 18973732]


Hoy D,Bain C,Williams G,March L,Brooks P,Blyth F,Woolf A,Vos T,Buchbinder R, A systematic review of the global prevalence of low back pain. Arthritis and rheumatism. 2012 Jun;     [PubMed PMID: 22231424]

Level 1 (high-level) evidence


Edlow JA. Managing Nontraumatic Acute Back Pain. Annals of emergency medicine. 2015 Aug:66(2):148-53. doi: 10.1016/j.annemergmed.2014.11.011. Epub 2015 Jan 9     [PubMed PMID: 25578887]


Maher C, Underwood M, Buchbinder R. Non-specific low back pain. Lancet (London, England). 2017 Feb 18:389(10070):736-747. doi: 10.1016/S0140-6736(16)30970-9. Epub 2016 Oct 11     [PubMed PMID: 27745712]


Hansen BB, Hansen P, Carrino JA, Fournier G, Rasti Z, Boesen M. Imaging in mechanical back pain: Anything new? Best practice & research. Clinical rheumatology. 2016 Aug:30(4):766-785. doi: 10.1016/j.berh.2016.08.008. Epub 2016 Nov 23     [PubMed PMID: 27931967]


Qaseem A, Wilt TJ, McLean RM, Forciea MA, Clinical Guidelines Committee of the American College of Physicians, Denberg TD, Barry MJ, Boyd C, Chow RD, Fitterman N, Harris RP, Humphrey LL, Vijan S. Noninvasive Treatments for Acute, Subacute, and Chronic Low Back Pain: A Clinical Practice Guideline From the American College of Physicians. Annals of internal medicine. 2017 Apr 4:166(7):514-530. doi: 10.7326/M16-2367. Epub 2017 Feb 14     [PubMed PMID: 28192789]

Level 1 (high-level) evidence


Cantrill SV, Brown MD, Carlisle RJ, Delaney KA, Hays DP, Nelson LS, O'Connor RE, Papa A, Sporer KA, Todd KH, Whitson RR, American College of Emergency Physicians Opioid Guideline Writing Panel. Clinical policy: critical issues in the prescribing of opioids for adult patients in the emergency department. Annals of emergency medicine. 2012 Oct:60(4):499-525. doi: 10.1016/j.annemergmed.2012.06.013. Epub     [PubMed PMID: 23010181]


Thackeray A, Hess R, Dorius J, Brodke D, Fritz J. Relationship of Opioid Prescriptions to Physical Therapy Referral and Participation for Medicaid Patients with New-Onset Low Back Pain. Journal of the American Board of Family Medicine : JABFM. 2017 Nov-Dec:30(6):784-794. doi: 10.3122/jabfm.2017.06.170064. Epub     [PubMed PMID: 29180553]


Arnold E, La Barrie J, DaSilva L, Patti M, Goode A, Clewley D. The Effect of Timing of Physical Therapy for Acute Low Back Pain on Health Services Utilization: A Systematic Review. Archives of physical medicine and rehabilitation. 2019 Jul:100(7):1324-1338. doi: 10.1016/j.apmr.2018.11.025. Epub 2019 Jan 24     [PubMed PMID: 30684490]

Level 1 (high-level) evidence


Chou R, Loeser JD, Owens DK, Rosenquist RW, Atlas SJ, Baisden J, Carragee EJ, Grabois M, Murphy DR, Resnick DK, Stanos SP, Shaffer WO, Wall EM, American Pain Society Low Back Pain Guideline Panel. Interventional therapies, surgery, and interdisciplinary rehabilitation for low back pain: an evidence-based clinical practice guideline from the American Pain Society. Spine. 2009 May 1:34(10):1066-77. doi: 10.1097/BRS.0b013e3181a1390d. Epub     [PubMed PMID: 19363457]

Level 1 (high-level) evidence


Atlas SJ, Deyo RA. Evaluating and managing acute low back pain in the primary care setting. Journal of general internal medicine. 2001 Feb:16(2):120-31     [PubMed PMID: 11251764]


Balagué F, Mannion AF, Pellisé F, Cedraschi C. Non-specific low back pain. Lancet (London, England). 2012 Feb 4:379(9814):482-91. doi: 10.1016/S0140-6736(11)60610-7. Epub 2011 Oct 6     [PubMed PMID: 21982256]


Shariat A, Cleland JA, Danaee M, Kargarfard M, Sangelaji B, Tamrin SBM. Effects of stretching exercise training and ergonomic modifications on musculoskeletal discomforts of office workers: a randomized controlled trial. Brazilian journal of physical therapy. 2018 Mar-Apr:22(2):144-153. doi: 10.1016/j.bjpt.2017.09.003. Epub 2017 Sep 6     [PubMed PMID: 28939263]

Level 1 (high-level) evidence


Tariq RA, George JS, Ampat G, Toney-Butler TJ. Back Safety. StatPearls. 2023 Jan:():     [PubMed PMID: 30085608]